Neuropeptides and inflammation: presumed mechanisms in neurogenic inflammation

Reference : Neuropeptides and inflammation: presumed mechanisms in neurogenic inflammation


	Document type :	Scientific journals : Article
	Discipline(s) :	Life sciences : Biochemistry, biophysics & molecular biology
	To cite this reference:	http://hdl.handle.net/10993/5738

Title :	Neuropeptides and inflammation: presumed mechanisms in neurogenic inflammation
Language :	English
Author, co-author :	Landry, Y. [> >]
	Bloch, J. G. [> >]
	Mousli, M. [> >]
	Bueb, Jean-Luc [University of Luxembourg > Faculty of Science, Technology and Communication (FSTC) > Life Science Research Unit >]
	Bronner, C. [> >]
Publication date :	1990
Journal title :	Pathologie et Biologie
Publisher :	Elsevier
Volume :	38
Issue/season :	1
Pages :	53-6
Peer reviewed :	Yes (verified by ORBi^lu)
ISSN :	0369-8114
e-ISSN :	1768-3114
City :	Paris
Country :	France
Keywords :	[en] Arthritis ; Humans ; Inflammation ; Mast Cells ; Membrane Proteins ; Neuropeptides ; Substance P ; physiopathology ; physiology
Abstract :	[en] Neuropeptides, among which substance P, VIP (Vasoactive intestinal peptide), somatostatin, neurotensin, dynorphin and enkephalins, are able to modulate inflammatory processes. Increasing interest is now devoted to these peptides in different inflammatory diseases, concerning skin, lung and joins. The effect of substance P can be dependent on its C-terminal moiety implicating by this way an interaction with specific neurokinin receptors or can be dependent on its N-terminal moiety which does not involve a specific membrane receptor. Such diversity of the action mechanisms of peptides should influence the evolution of the anti-inflammatory therapeutic.
Permalink :	http://hdl.handle.net/10993/5738

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